Bumetanide induces post-traumatic microglia-interneuron contact to promote neurogenesis and recovery
La bumétanide induit un contact microglie-interneurone post-traumatique pour promouvoir la neurogenèse et la récupération
Résumé
Although the Na-K-Cl cotransporter (NKCC1) inhibitor bumetanide has prominent positive effects on the pathophysiology of many neurological disorders, the mechanism of action is obscure. Attention for elucidating the role of Nkcc1 has been mainly focused on neurons. Recent single cell
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